Noressa

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• Noressa burned 482.76 calories doing Elliptical trainer: general.

  An elliptical trainer (also cross trainer) is a stationary exercise... read more at The Daily Plate.

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• Noressa is now friends with becmac79

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• Noressa posted For those worried about cholesterol in the group For those worried about cholesterol
  “  http://waroninsulin.com/nutrition/the-straight-dope-on-cholesterol-part-i   Read this series.  He's just posted the final update today.  There is A LOT of reading.  There is a lot to understand.  And ultimately, the takeaways should be eating cholesterol DOES NOT increase your cholesterol.  DO NOT WORRY ABOUT WHAT YOU INGEST.   * Cholesterol is “just” another fancy organic molecule in our body but with an interesting distinction: we eat it, we make it, we store it, and we excrete it – all in different amounts. * The pool of cholesterol in our body is essential for life.  No cholesterol = no life. * Cholesterol exists in 2 forms – unesterified or “free” (UC) and esterified (CE) – and the form determines if we can absorb it or not, or store it or not (among other things). * Much of the cholesterol we eat is in the form of CE. It is not absorbed and is excreted by our gut (i.e., leaves our body in stool). The reason this occurs is that CE not only has to be de-esterified, but it competes for absorption with the vastly larger amounts of UC supplied by the biliary route. * Re-absorption of the cholesterol we synthesize in our body (i.e., endogenous produced cholesterol) is the dominant source of the cholesterol in our body. That is, most of the cholesterol in our body was made by our body. * The process of regulating cholesterol is very complex and multifaceted with multiple layers of control.  I’ve only touched on the absorption side, but the synthesis side is also complex and highly regulated. You will discover that synthesis and absorption are very interrelated. * Eating cholesterol has very little impact on the cholesterol levels in your body. This is a fact, not my opinion.  Anyone who tells you different is, at best, ignorant of this topic.  At worst, they are a deliberate charlatan. Years ago the Canadian Guidelines removed the limitation of dietary cholesterol. The rest of the world, especially the United States, needs to catch up.   * Cholesterol and triglycerides are not soluble in plasma (i.e., they can’t dissolve in water) and are therefore said to be hydrophobic. * To be carried anywhere in our body, say from your liver to your coronary artery, they need to be carried by a special protein-wrapped transport vessel called a lipoprotein. As these “ships” called lipoproteins leave the liver they undergo a process of maturation where they shed much of their triglyceride “cargo” in the form of free fatty acid, and doing so makes them smaller and richer in cholesterol. * All lipoproteins are part of the human lipid transportation system and work harmoniously together to efficiently traffic lipids. * The measurement of cholesterol has undergone a dramatic evolution over the past 70 years with technology at the heart of the advance. Currently, most people in the United States (and the world for that matter) undergo a“standard” lipid panel, which only directly measures TC, TG, and HDL-C.  LDL-C is measured or most often estimated. More advanced cholesterol measuring tests do exist to directly measure LDL-C (though none are standardized), along with the cholesterol content of other lipoproteins (e.g., VLDL, IDL) or lipoprotein subparticles. *The most frequently used and guideline-recommended test that can count the number of LDL particles is either apolipoprotein B or LDL-P NMR, which is part of the NMR LipoProfile.  NMR can also measure the size of LDL and other lipoprotein particles, which is valuable for predicting insulin resistance in drug naïve patients, before changes are noted in glucose or insulin levels. The progression from a completely normal artery to a “clogged” or atherosclerotic one follows a very clear path: an apoB containing particle gets past the endothelial layer into the subendothelial space, the particle and its cholesterol content is retained, immune cells arrive, an inflammatory response ensues “fixing” the apoB containing particles in place AND making more space for more of them. *While inflammation plays a key role in this process, it’s the penetration of the endothelium and retention within the endothelium that drive the process. The most common apoB containing lipoprotein in this process is certainly the LDL particle. However, Lp(a) and apoB containing lipoproteins play a role also, especially in the insulin resistant person. *If you want to stop atherosclerosis, you must lower the LDL particle number. Period.”

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• Noressa posted "The injury and inflammation in our blood vessels is caused by the low fat diet recommended for years by mainstream medicine." in the group "The injury and inflammation in our blood vessels is caused by the low fat diet recommended for years by mainstream medicine."
  “  http://www.preventdisease.com/news/12/030112_World-Renown-Heart-Surgeon-Speaks-Out-On-What-Really-Causes-Heart-Disease.shtml?source=Patrick.net”